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This b7-33 peptide has a molecular structure that allows it to bind to the Relaxin Family Peptide Receptor 1 (RXFP1) and preferentially activate the pERK pathway over the cAMP pathway, which is often associated with tumorigenesis. When you purchase b7-33 peptide for sale from us, you receive lyophilized powder in sterile vials, verified by third-party laboratories to high purity standards. Researchers seeking b7-33 peptide for sale appreciate our transparent documentation, including batch-specific certificates of analysis.

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What is B7-33

B7-33 is a rationally designed single-chain peptide derived from the B-chain of human relaxin-2. This B7-33 peptide was developed through extensive structure-activity relationship studies to identify the minimal structure required to ensure binding and activation of the Relaxin Family Peptide Receptor-1 (RXFP1). What makes B7-33 unique is that it reproduces the beneficial anti-fibrotic effects of relaxin without triggering pathways often associated with tumorigenesis.

This B7-33 peptide has a positive charge, improved half-life compared to native relaxin, and is water-soluble, making it a more practical option for research applications. For more information on what is b7-33, search Google or Bing.

B7-33 Anti Fibrosis

B7-33 anti fibrosis research has demonstrated significant potential in modulating fibrotic signaling pathways. Studies have shown that B7-33 inhibits the excessive Transforming Growth Factor β-1 (TGFβ-1) signaling, thereby reducing pro-fibrotic effects on myofibroblast differentiation and aberrant extracellular matrix production, which is the root of fibrosis.

The b7-33 anti fibrosis mechanism involves binding to RXFP1 and promoting ERK1/2 phosphorylation, which downregulates the expression of collagen type I and other pro-fibrotic genes. In hypertrophic scar fibroblast studies, B7-33 demonstrated greater collagen type I α-1 chain downregulation than reference antifibrotic agents. This b7-33 anti fibrosis activity has been validated in numerous preclinical animal studies.

B7-33 Peptide

B7-33 peptide is a synthetic 33-amino acid single-chain peptide that functions as a functionally selective agonist of the G protein-coupled receptor, RXFP1. This B7-33 peptide was designed to overcome the limitations of native relaxin-2, which has a complex two-chain, three-disulfide-bonded structure that is difficult and costly to synthesize.

The B7-33 peptide retains the anti-fibrotic, anti-inflammatory, and vasodilatory effects of the native hormone. In vitro studies have shown that the B7-33 peptide is biocompatible with both normal and hypertrophic human fibroblasts, maintaining >70% viability after 72 hours of exposure. For B7-33 peptide research, choose Elite Peptides Supply. Visit our homepage to learn more about B7-33 peptide.

B7 33 Protein

B7 33 protein refers to the peptide’s classification as a small protein or peptide. The b7 33 protein is a synthetic analogue of the relaxin hormone, which is a member of the insulin-like peptide family. Unlike native relaxin, which is a two-chain protein with disulfide bonds, the b7 33 protein is a single-chain linear peptide that retains potent beneficial agonistic effects. The b7 33 protein has a molecular weight and amino acid sequence that allows it to bind to RXFP1 with high affinity and specificity. For information on b7 33 protein research, search Google Scholar.

B7 33 Benefits for Men

Research into b7 33 benefits for men has been documented in cardiovascular and fibrosis studies. The b7 33 benefits for men include potential applications in cardiomyopathy, where it has been shown to reduce left ventricular inflammation, fibrosis, and cardiomyocyte hypertrophy. The b7 33 benefits for men also extend to vascular function, as equimolar doses of B7-33 replicated the acute beneficial vascular effects of serelaxin in rat mesenteric arteries.

Additionally, b7 33 benefits for men may include organ-protective effects through its ability to inhibit fibrotic signaling pathways. For comprehensive b7 33 benefits for men information, consult primary literature.

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Benefits of b7 33 in research models are extensive and well-documented. The primary benefits of b7 33 include anti-fibrotic effects, where it has been shown to downregulate collagen type I expression and modulate profibrotic gene expression. The benefits of b7 33 also encompass cardioprotective actions, including reducing infarct size, preserving cardiac function, and normalizing left ventricular inflammation.

Additionally, the benefits of b7 33 include vasoprotective functions, such as enhancing endothelium-dependent relaxation and preventing endothelial dysfunction. The benefits of b7 33 also extend to preeclampsia research, where it has been shown to lower blood pressure and proteinuria in rodent models. For a full list of benefits of b7 33, search Google Scholar.

B7 33 Dosage

Determining b7 33 dosage for research requires consulting published literature for your specific model. Studies examining b7 33 dosage in mice used 0.25 mg/kg/day subcutaneous for cardiomyopathy models. For cardioprotection studies, b7 33 dosage of 50 and 100 nmol/L were used in vitro. The b7 33 dosage for fibrosis research in hypertrophic scar fibroblasts used concentrations of 0.03–0.3 μg/mL.

For preeclampsia research, b7 33 dosage of 35 μg i.p. biweekly was used in rodent models. When establishing b7 33 dosage, always start with the lowest effective concentration. For assistance with b7 33 dosage, contact our research support team.

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Our b7 33 peptide can be formulated as b7 33 peptide nasal spray for research applications. For b7 33 peptide nasal spray protocols, consult published literature. Use Google to find b7 33 peptide nasal spray research.

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B7 33 peptide dosage for research varies by model and application. Studies examining b7 33 peptide dosage in cardiac research used 0.25 mg/kg/day. The b7 33 peptide dosage for fibrosis studies in vitro used concentrations of 0.03–0.3 μg/mL . In preeclampsia research, b7 33 peptide dosage of 35 μg i.p. biweekly was used. The b7 33 peptide dosage for vascular studies in rats used 13.3 μg/kg as an acute bolus injection.

When establishing b7 33 peptide dosage, consult primary literature for your specific model. For assistance with b7 33 peptide dosage, contact our research support team.

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Q2: What is B7-33 peptide?
A2: B7-33 is a synthetic single-chain peptide analogue of human relaxin-2 that acts as a functionally selective agonist of RXFP1. This B7-33 peptide retains the anti-fibrotic effects of relaxin without triggering tumorigenic pathways.

Q3: What are the benefits of B7-33 in research?
A3: Benefits of b7 33 include anti-fibrotic effects, cardioprotection, and vasoprotection. B7-33 has shown significant reduction in collagen type I expression and left ventricular fibrosis.

Q4: What is the B7-33 peptide dosage for research?
A4: B7-33 peptide dosage varies: 0.25 mg/kg/day SC in mice, 0.03–0.3 μg/mL in vitro, and 35 μg i.p. biweekly in preeclampsia models. Consult literature for b7 33 peptide dosage guidance.

Q5: Can I buy B7-33 peptide for research in Canada?
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Q6: What is the mechanism of B7-33 anti fibrosis?
A6: B7-33 anti fibrosis involves binding to RXFP1 and promoting ERK1/2 phosphorylation, which inhibits TGF-β1 signaling. B7-33 downregulates collagen type I and other pro-fibrotic genes.

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Q8: What is the difference between B7-33 and relaxin?
A8: B7-33 is a single-chain peptide analogue of relaxin that is easier to synthesize. B7-33 replicates the beneficial effects of relaxin without triggering tumor-associated pathways.

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